
RASFF – norovirus (GII /2g) in live oysters from France in Italy
RASFF – norovirus (GI, GII /2g) in live oysters from France in Italy
RASFF – norovirus (GI and GII /g) in live oysters (Crassostrea Gigas) from France in Italy

RASFF – foodborne outbreak suspected (norovirus) to be caused by live oysters from France in the Netherlands
Posted in food bourne outbreak, food contamination, Food Hygiene, Food Illness, Food Inspections, Food Micro Blog, Food Microbiology Blog, Food Microbiology Testing, Food Pathogen, food recall, Food Safety, Food Safety Alert, Food Testing, Food Virus, foodborne outbreak, foodbourne outbreak, Norovirus, outbreak, RASFF, Uncategorized, Virus

RASFF – ochratoxin A (37.66 µg/kg – ppb) in dried figs from Turkey in Germany

RASFF – enteropathogenic Escherichia coli (stx- eae+ /25g) in organic leek seeds for sprouting from China, via Italy in Norway
Posted in E.coli, eae, food contamination, Food Hygiene, Food Inspections, Food Micro Blog, Food Microbiology, Food Microbiology Blog, Food Microbiology Testing, Food Pathogen, Food Poisoning, food recall, Food Safety, Food Safety Alert, Food Testing, Food Toxin, RASFF, STEC, STEC E.coli, STX 1, STX 2, Uncategorized

RASFF – Salmonella enterica ser. Typhimurium (presence /25g) in chilled turkey meat from Poland in Poland
RASFF – Salmonella (presence /25g) in frozen half chicken breast innerfillet from Brazil in the UK
RASFF – Salmonella enterica ser. Typhimurium (present /25g) in frozen veal and turkey doner from Germany in Germany
RASFF – Salmonella enterica ser. Infantis (presence /25g) in chicken wings from Poland in Lithuania
RASFF – Salmonella enterica ser. Enteritidis (in 1 out of 5 samples /25g) in chilled chicken quarters from Poland in Lithuania
RASFF – Salmonella (presence /25g) in frozen kebab from Slovenia in Italy
RASFF – Salmonella enterica ser. Moroto in whitish sesame seeds from Sudan in Greece

Image CDC
In this study, it was aimed to design the time‐predicted inactivation model for various growth curves and the growth parameters representing the changes in the inactivation of Staphylococcus aureus in probiotic white cheese. For this purpose, S. aureus were inoculated in the pasteurized milk and the production of probiotic white cheese was performed in five different groups, and then samples were matured for 60 days at 4°C. S. aureus, Lactobacillus rhamnosus, Lactobacillus casei Shirota, and mesophilic starter culture levels of the samples were analyzed on the 1st, 4th, 7th, 10th, 15th, 30th, and 60th days of maturation. The inactivation model evaluation was performed by using the number of microorganisms counted with Fourier modeling method. In determining the model to be applied, the coefficient of determination (R2) and the sum of squared errors (SSE) for the mean number of microorganisms detected via modeling method were used. As conclusions, the number of S. aureus has decreased by approximately 5 logarithmic units at the end of the storage period by using probiotics together with the starter culture. It was determined that the Fourier modeling method is a suitable inactivation model with R2 values of 0.9847–0.9986 and SSE values of 0.094–0.510.
Efficient Laboratories is voluntarily recalling one lot each of Rompe Pecho EX, CF, and MAX liquid. These products are used to treat symptoms of the flu and the common cold. The three lots have been found to contain microbial contamination.
The recall notice did not state what type of microbe may have contaminated these products, but it did state that “In rare circumstances, consumption of Rompe Pecho from these lots could result in vomiting and diarrhea. Efficient Laboratories has not received any reports of adverse events to date.”
Contamination of edible produce leaves with human bacterial pathogens has been associated with serious disease outbreaks and has become a major public health concern affecting all aspects of the market, from farmers to consumers. While pathogen populations residing on the surface of ready-to-eat produce can be potentially removed through thorough washing, there is no disinfection technology available that effectively eliminates internal bacterial populations. By screening 303 multi-gene deletion (MGD) mutants of Salmonella enterica serovar Typhimurium (STm) 14028s, we were able to identify ten genomic regions that play a role in opening the stomatal pore of lettuce leaves. The major metabolic functions of the deleted regions are associated with sensing the environment, bacterium movement, transport through the bacterial membrane, and biosynthesis of surface appendages. Interestingly, at 21 days post inoculation, seven of these mutants showed increased population titers inside the leaf, two mutants showed similar titers as the wild type bacterium, whereas one mutant with a large deletion that includes the Salmonella pathogenicity island 2 (SPI-2) showed significantly impaired persistence in the leaf apoplast. These findings suggest that not all the genomic regions required for initiation of leaf colonization (i.e., epiphytic behavior and tissue penetration) are essential for continuing bacterial survival as an endophyte. We also observed that mutants lacking either SPI-1 (Mut3) or SPI-2 (Mut9) induce callose deposition levels comparable to those of the wild type STm 14028s; therefore, these islands do not seem to affect this lettuce defense mechanism. However, the growth of Mut9, but not Mut3, was significantly impaired in the leaf apoplastic wash fluid (AWF) suggesting that the STm persistence in the apoplast may be linked to nutrient acquisition capabilities or overall bacterial fitness in this niche, which are dependent on the gene(s) deleted in the Mut9 strain. The genetic basis of STm colonization of leaves investigated in this study provides a foundation from which to develop mitigation tactics to enhance food safety.
When food makes people sick, some blame birds because they hang around farms, and their feces can contain E. coli, Salmonella and Campylobacter, three common pathogens that can cause food-borne illness.
But a recent Washington State University study published in Biological Reviews on Jan. 31 has found scant evidence to support the link between wild birds and human illness involving those three pathogens.
The perceived risk of wild birds can impact their survival, said Olivia Smith, lead author on the study and a recent WSU Ph.D. graduate.
“Farmers are being encouraged to remove wild bird habitat to make their food safer, but it doesn’t appear that these actions are based on data,” Smith said. “When you restrict birds from agricultural settings, you are doing something that can lead to their decline.”
Bird populations have been falling rapidly in recent decades. Scientists estimate that since 1970, North America has lost more than three billion birds. In light of this, the WSU researchers highlighted the need for more definitive research before destroying habitat and banning birds from fields in the name of food safety.